The strategy paid off.Kuo and his colleagues? Examination of the postnatal brain of mice at autopsy seven and 14 days after the genetic modification revealed that the absence of proteins and Numb Numblike led to severe brain ventricle enlargement. This defect resulted from damage of ependymal cells however, epithelial lining of the cerebral ventricles, of neuroblasts that develop into neurons. Thus it was evident, as in embryonic mouse, the proteins. A crucial role in the maintenance of cells in the region, and revealed a hitherto unknown function for them in assuring ependymal integrity.
– Two central questions for scientists exploring the potential of these cells to treat disease , but little is known about the molecular programs that neural neural stem cells in the postnatal mouse, or how neural stem cells may respond to tissue damage is known.Thyroid cancer in remote locations to distant sites with a poor prognosis, rather an experimental drug that inhibits tumor blood vessels may to slow progression of the disease in some patients a research team around detectives leading the University of Texas MD Anderson trusted in the 3 July issue of the New England Journal of Medicine.
The program usually has around 80 period I clinical trials ongoing, drug that of targeted therapies first time in cancer patients. Addition to closely monitoring a new drug for safety, it test the program compared different types of cancer. The important to of molecular targets potential new medicines on cancer in general, but at this stage to medicines are not specific to any particular kind of cancer, so we are the patient with various kinds at these early studies, short skirt told. This will give us an unique reply signals response signals first time in the some cancers, flagging a drug for further extensive study in phase of II or phase III clinical trials. .