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Scientists discovered that when ApoE4 exists, the brain is less efficient at ridding itself of the toxic materials, because a molecule that is much slower at getting rid of the substance becomes a lot more involved. The brand-new results are in line with a body of analysis amassed during the last 15 years by the leader of the team, Berislav Zlokovic, M.D., Ph.D., of the University of Rochester INFIRMARY, that blood circulation has a key role in the condition. His group has identified a lot of the molecular machinery that allows amyloid-beta to sidestep the body’s safeguards and enter the mind, and he has found out molecules that falter when the toxic protein accumulates in the brain.